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IJMS | Free Full-Text | Stress-Induced Transcriptomic Changes in Females with Myalgic Encephalomyelitis/Chronic Fatigue Syndrome Reveal Disrupted Immune Signatures
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Frontiers | Aberrations in the Cross-Talks Among Redox, Nuclear Factor-κB, and Wnt/β-Catenin Pathway Signaling Underpin Myalgic Encephalomyelitis and Chronic Fatigue Syndrome
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(PDF) Increased nuclear factor-κB and loss of p53 are key mechanisms in Myalgic Encephalomyelitis/chronic fatigue syndrome (ME/CFS) | Gerwyn Morris - Academia.edu
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The nuclear factor kappa B signaling pathway: integrating metabolism with inflammation: Trends in Cell Biology
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The inflammatory and oxidative and nitrosative (IO&NS) pathophysiology... | Download Scientific Diagram
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Frontiers | Aberrations in the Cross-Talks Among Redox, Nuclear Factor-κB, and Wnt/β-Catenin Pathway Signaling Underpin Myalgic Encephalomyelitis and Chronic Fatigue Syndrome
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PDF] Why myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) may kill you: disorders in the inflammatory and oxidative and nitrosative stress (IO&NS) pathways may explain cardiovascular disorders in ME/CFS. | Semantic Scholar
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Changes in the transcriptome of circulating immune cells of a New Zealand cohort with myalgic encephalomyelitis/chronic fatigue syndrome - Eiren Sweetman, Margaret Ryan, Christina Edgar, Angus MacKay, Rosamund Vallings, Warren Tate, 2019
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CHRONIC FATIGUE SYNDROME, MITOCHONDRIAL DYSFUNCTION AND PARASYMPATHETIC EXCESS - Dr. Nicholas L. DePace, M.D., F.A.C.C.
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Increased nuclear factor-κB and loss of p53 are key mechanisms in Myalgic Encephalomyelitis/chronic fatigue syndrome (ME/CFS) - ScienceDirect
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Impaired calcium mobilization in natural killer cells from chronic fatigue syndrome/myalgic encephalomyelitis patients is associated with transient receptor potential melastatin 3 ion channels - Nguyen - 2017 - Clinical & Experimental Immunology ...
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